kibbles said:
prodigalfan said:
a poster at an mj board is typing the testimony of dr. shafer, and below is what s/he wrote:
between 9-10am his breathing is slowing. carbon dioxide levels would be going up and that keeps u breathing. at 10am carbon levels are higher but hes still breathing as hes on the oxygen. murray would be thinking mj is fine and asleep. as u get to near noon the carbon cant keep him breathing any longer. so the breathing slows can the slow breathing be detected? yes with caponomentry and the pulse oxy. if he was sat next to mj he would have seen the breathing slow.easily saved mj if murray was there
so at 11-11.45 whats happening? breathing slower and slower interruptions in mjs breathing mj may have made a noise like gasping/coughing. might not have though. shaffer says the infusion was still running when mj died and thats why the concentrations were so high. its the only senario he could come up with that would produce such a level and thats based off murrays own words and the amount of diprivan he brought.fits with murray saying that he left mj alone. he observed mj for an hour that everything was ok but it wasnt cause there was no pulse oxy carbon monitoring. shaffer says all the data in this case fits perfectly with this senario. theres no data that doesnt fit. shaffer says this in his opinion is what happened to mj .
what if the diprivan runs out at noon and mj is still alive? the concentrations would drop quickly like he said b4. so he believes the infusion was still running when mj died because the levels never dropped they were still high and the diprivan kept going into mj. man this is hard *fills up*
shaffer is gonna show how u set up a IV infusion. chernoff gettting pissed saying this is based off a hypothetical .no its based off shaffers testimony. all those hypotheticals are coming back to haunt chernoff
so even though you may disagree, the doctor says that it is possible that a coughing noise could have come from mj, though obviously we have no way of knowing.
Of course I was not there with MJ. I am just tellling you my experience recovering patients on Propofol.
If they take a breath and there is some blocking of the airway either the lungs will expand and contract like taking a breath but no air goes in because the windpipe is closed off. If the windpipe is only half closed off so some air IS going into the windpipe the noise you will hear from the patient is a very LOUD snoring noise.
You know, when a person snores that is exactly what is happening. They are breathing and the fleshy part of the mouth is so relaxed it gets in the way of the airflow and the noise is the air vibrating against the fleshy tissues that then vibrate against the vocal cords.
when you have sleep apnea (you know people who snore and then stop breathing for a few seconds and they wake up hundred of times per night) is caused when the fleshy part of the mouth totally closes off the airway and the person stops breathing.
But because they are only asleep and not unconcious, the person WAKES UP and moves head/neck in such a way that the windpipe is uncovered and normal breathing resumes.
That is why people who are unconcious are so vunerable. That is why you NEVER leave a person unattended. That is why you have at least 2 people watching just in case one person needs to go to bathroom, or they fall asleep or they themselves have a medical emergency. Someone who is unconcous can't even clear their own throat. They can't even do a slight adjustment to their head so that their windpipe is clear.
"Remember, one man's filler is another man's killer" -- Haystack |